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观察26例施行晶体囊外摘出联合植入后房型人工晶体的白内障患者,球后麻醉10分钟后,视力均明显下降。认为其机理可能是:(1)麻醉剂对视神经的作用;(2)麻醉剂压迫致使视神经缺血;(3)麻醉剂压迫导致内视,致使感光细胞成分和视网膜色素上皮出现饱和现象。所有患者麻醉后上斜肌仍有残余功能,7例麻醉后内直肌能轻微运动,这可能与药物未进入肌锥、药效已经减弱、局部对药物有较强的抵抗力或压迫眼眶时间不充分等有
26 cases of cataract patients who underwent extracapsular lens extraction combined with posterior chamber intraocular lens implantation were observed. Visual acuity was significantly decreased after 10 minutes of retrobulbar anesthesia. The mechanism is believed to be: (1) the effects of anesthetics on the optic nerve; (2) oppression of the optic nerve caused by compression of the anesthetic; and (3) oppression of the anesthetic causes endoscopy, causing saturation of photoreceptor cell components and the retinal pigment epithelium. All patients with upper oblique anesthesia after anesthesia still residual function, 7 cases of rectus muscle anesthesia after a slight exercise, which may not enter the muscle cones medicine, efficacy has weakened, the local strong resistance to drugs or oppression of orbital time Not full waiting