ERK5 MEDIATES ADVANCED GLYCATION END PRODUCTS-INDUCED IMPAIRMENT OF ANGIOGENIC CAPACITY IN CARDIAC E

来源 :第二十四届长城国际心脏病学会议、亚太心脏大会暨国际心血管病预防与康复会议(The 24th Great Wall Int | 被引量 : 0次 | 上传用户:yannini01
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Objectives Diabetes-induced impairment of collateral formation in ischaemic myocardium has been demonstrated both in experimental and clinical studies, which may contribute to unfavorableprognosis among diabetic individuals.However, the underlying mechanism remains to be defined.Endothelial cell (EC) is thought to be one of the important elements in the development and maturation of coronary collaterals.
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