Chemotherapy and EGFR-tyrosine Kinase Inhibitors in The Treatment of Non-small Cell Lung Cancer with

来源 :2013抗肿瘤药物研究新进展与肿瘤个性化药物治疗论坛 | 被引量 : 0次 | 上传用户:liongliong519
下载到本地 , 更方便阅读
声明 : 本文档内容版权归属内容提供方 , 如果您对本文有版权争议 , 可与客服联系进行内容授权或下架
论文部分内容阅读
  [Purpose] Chemotherapy and epidermal growth factor receptor tyrosine kinase inhibitor(EGFR-TKIs) treatments remain under dispute for patients with brain metastases from non-small cell lung cancer(NSCLC).[Methods] We retrospectively studied the outcomes of solely localized treatments or localized treatments in combination with chemotherapy and/or EGFR-TKIs medications for 210 NSCLC patients with brain metastases.The effects of treatment modality, Karnofsky performance status(KPS), age, primary tumor histology, number of brain metastases and other factors on survival were analyzed and the robustness of two prognostic indices (the RPA and GPA models) were evaluated.[Results] The median survival time (MST) for patients with systemic medication and localized treatments was higher than that with localized treatments alone (11.0 vs 3.0 months,P=0.000).Within the systemic medication group,the MST was significantly higher for EGFR-TKI than that for chemotherapy treatments (12.0 vs 9.0 months, P=0.002).The MST of patients with mutated EGFR gene was 20 months vs 8 months for patients with the EGFR wild type gene in the EGFR-TKI group.The MST with pemetrexed medication was significantly higher than that with other chemotherapies (13.0 vs 7.0 months,P=0.006).In a multivariate analysis,prognosis was significantly correlated with treatment modality (P=0.000),KPS (P=0.000), number of brain metastases (P=0.001) and tumor histological type (P=0.007).In the Graded Prognostic Assessment (GPA) model, the survival curves of subgroups showed clear separations.[Conclusions] NSCLC patients with brain metastases had a longer survival benefit from chemotherapies, especially pemetrexed and/or TKIs along with localized treatments and the GPA index is a robust model for prognostic evaluation.
其他文献
目的:本研究旨在探讨胃肠道间质瘤imatinib继发性耐药机制和砷剂联合沙利度胺对imatinib耐药的GIST的抗肿瘤作用.方法:建立imatinib耐药的人GIST细胞系及其裸鼠移植瘤模型.体外实验,MTT法检测sunitinib、砷剂(三氧化二砷,As2O3)、沙利度胺(Thai)等药物对imatinib耐药的人GIST细胞系的体外药敏实验.Western blot法检测imatinib耐药
目的:研究浙产西红花的体内外抗癌活性和可能机制.方法:以伊朗西红花作为参照,选取浙江建德和湖州两地西红花为研究对象.MTT法测定西红花对人非小细胞肺癌细胞株A549和人小细胞肺癌细胞株H446增殖活性的抑制作用,Annexin-V/PI双染法检测西红花的促凋亡效应,Hoechst染色观察凋亡细胞核形态改变.西红花提取液100mg/kg/d灌胃28天后,观察裸鼠皮下移植瘤的生长情况,检测肿瘤组织ca
[Purpose]-To clear the optimal time of concurrent thoracic radiotherapy (rRT) with etoposide and cisplatin/carboplatin (EP/EC) chemotherapy for limited-disease small cell lung cancer (LD SCLC).[Materi
会议
Purpose: Combined small cell lung cancer (CSCLC) is currently considered a subset of SCLC and have been reported to account for less than 1%~3.2% of all SCLCs.Accurate understanding of CSCLCs is of gr
会议
目的:本实验通过研究Dasatinib对AML细胞的诱导分化作用,并进一步探讨自噬在这一诱导分化作用中所扮演的角色,为发现新型分化诱导剂提供研究方向和理论依据.方法:用台盼蓝拒染法结合血细胞计数观察细胞增殖情况;CD11b结合流式细胞术,NBT还原染色法以及瑞士吉姆萨染色法测定细胞分化;PI/AnnexinV染色结合流式细胞术检测细胞凋亡;PI染色结合流式细胞术确定细胞周期分布;MDC染色考察细胞
目的:建立临床药师在临床药物治疗过程中的药学服务实践工作模式,为提高中晚期癌症患者癌痛治疗提供参考.方法:临床药师参与1例晚期胃癌患者止痛治疗,及时发现治疗中需注意的问题,提出合理建议.结果:临床药师建议的药物治疗方案被部分采纳,使患者疼痛症状缓解,不良反应减轻.结论:临床药师通过参与临床镇痛药物的治疗,对患者个体化和规范化治疗起到了积极的作用.
会议
[Purpose] Nasopharyngeal carcinoma (NPC) is the most common malignant tumor in Southeast Asia and radio-resistance remains a serious obstacle to successful treatment.Curcumin has been used to treat NP
会议
会议