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In this study the effect of electric stimulus at auricular visceral points on the rabbit’s myoelectric activity of sphincter of oddi(SO)under three conditions(normal,celiac vagal trunk sectioned and M-receptor blocked)was observed. A needle electrode was poisitioned in SO to record myoelectric activity. The electric stimulus frequency 10 hertz, intensity 4～6 volts and time 10 minutes. In 14 animals the change of myoelectric activity of SO responding to auricular point stimulus was recorded before and after vagotomy, respectively. Results: under normal condition(vagus not amputated)after stimulus the SO myoelectric activity rate decreased from the control level 7.17±1.84 spike bursts/min(SB/min)to 4.76±1.68 SB/min(p<0.01),but in vagotomy background after stimulus the SO myoelectric activity rate slightly decreased from 4.14±1.57 SB/min to 3.36±1.60 SB/min.(p>0.05).In another 15 animals atropine(0.2mg/kg)was administrated intravenously to block M-receptor. In Vagotomy group auricular point stimulus caused SO myoelectric activity rate to fall from the control level 8.19±1.29 SB/min to 4.71±1.29 SB/min(p<0.01).On the condition of M-receptor blocked the rate altered from 4.15±1.10 SB/min to 4.25±0.92 SB/min(p>0.05).All these results indicate:①Electric stimulus of auricular visceral points has
In this study the effect of electric stimulus at auricular visceral points on the rabbit’s myoelectric activity of sphincter of oddi (SO) under three conditions (normal, celiac vagal trunk sectioned and M-receptor blocked) was observed. A needle electrode was poisitioned in SO To record myoelectric activity. The electric stimulus frequency 10 hertz, intensity 4 ~ 6 volts and time 10 minutes. In 14 animals the change of myoelectric activity of SO responding to auricular point stimulus was recorded before and after vagotomy, respectively. Results: under normal condition (vagus not amputated) after stimulus the SO myoelectric activity rate decreased from the control level 7.17 ± 1.84 spike bursts / min (SB / min) to 4.76 ± 1.68 SB / min In another 15 animals atropine (0.2 mg / kg) was administrated intravenously to block M-receptor. In Vagotomy group (0.2 mg / kg) was significantly reduced from 4.14 ± 1.57 SB / min to 3.36 ± 1.60 SB / auricul ar point stimulus caused SO myoelectric activity rate to fall from the control level 8.19 ± 1.29 SB / min to 4.71 ± 1.29 SB / min (p <0.01) .On the condition of M-receptor blocked the rate altered from 4.15 ± 1.10 SB / min to 4.25 ± 0.92 SB / min (p> 0.05) .All these results indicate: ①Electric stimulus of auricular visceral points has