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环—磷酸腺苷(cAMP)系一种核苷酸,存在于各种哺乳类动物的组织和细胞外液中。目前,认为cAMP是激素效应的“辅助传递者”。在实验性心肌缺血中,心肌细胞内液的cAMP增多,推测系由于内源性儿茶酚胺释放并激活腺苷环化酶所致。这种现象被认为是一种对冠状动脉血流量减少的早期生物化学反应。急性心肌梗塞时,血浆儿茶酚胺和血清皮质醇的水平均有增多,儿茶
CAMP is a nucleotide found in the tissues and extracellular fluids of various mammalian species. Currently, cAMP is considered a “helper” of the hormonal effect. In experimental myocardial ischemia, intracellular cardiomyocyte cAMP increased presumably due to the release of endogenous catecholamines and activation of adenosine cyclase caused. This phenomenon is thought to be an early biochemical reaction to reduced coronary blood flow. Acute myocardial infarction, plasma catecholamines and serum cortisol levels have increased, catechu