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以前的研究发现亲脂性过氧化物可诱发小鼠短暂性脑缺血血样发作（TIA-发作），本研究通过观察亲脂性过氧化物叔丁基氢过氧化物（t-BHP）的化学结构类似物叔丁醇（t-BA）及过氧化氢（H2O2）盐水溶液和脂肪乳溶液在诱发小鼠TIA-发作中的作用，主要探讨过氧化物诱发小鼠TIA-发作的化学基础。结果如下：t-BHP（0．11mol／L）可诱发小鼠TIA-发作，叔丁醇（t-BA，0.135mol／L）不能诱发小鼠TIA-样发作，H2O2盐水溶液也不能诱发小鼠TIA-发样作，但H2O2脂肪乳溶液可诱发小鼠TIA-样发作：抗氧化剂VitC＋CitE可抑制血液内过氧化物的作用。因此，我们认为过氧化物诱发小鼠脑缺血样发作的化学结构基础是过氧基（-OOH），其中过氧化物的亲脂性起关键作用。
Previous studies have found that lipophilic peroxides induce episodes of transient ischemic blood in mice (TIA-seizures). This study was conducted by observing the chemical structural analogues of t-BHP, a lipophilic peroxide (T-BA) and hydrogen peroxide (H2O2) saline solution and fat emulsion in induced TIA-induced seizures in mice, mainly to investigate the chemical basis of peroxide-induced TIA-seizures in mice. The results were as follows: TIA-induced seizures were induced by t-BHP (0.11mol / L) and TIA-like seizures could not be induced by t-BA (0.135mol / L) Mouse TIA-hair samples, but H2O2 fat emulsion solution can induce mouse TIA-like seizures: Antioxidant VitC + CitE can inhibit the role of blood peroxide. Therefore, we believe that the chemical structure of peroxide-induced cerebral ischemia-like episodes in mice is based on peroxide (-OOH), in which lipophilicity of peroxide plays a key role.