论文部分内容阅读
Objective: We report a clinical description of pure sensory stroke based on da ta collected from a prospective acute stroke registry. Methods: From 2500 acute stroke patients includedin a hospital-based prospective stroke registry over a 12-year period, 99 were identified as having pure sensory stroke. Results:Pure sensory stroke accounted for 4.7%of all acute stroke patients, 5.4%of acute is chemic stroke, and 17.4%of lacunar syndromes. Complete hemisensory syndrome was present in 80 patients and incomplete hemisensory syndrome in 19 (cheirooral sy ndrome 12, cheiro-oral-pedal 6, isolated oral syndrome1). The lacunar hypothes is was fulfilled in 88%of patients.Atherothrombotic infarction occurred in 8 pa tients, intracerebral hemorrhage in 3, and stroke of undetermined cause in 1.Hem orrhagic pure sensory stroke was diagnosed in 1%of all cases of hemorrhagic str oke (n=270) in the database. Outcome was good (in-hospital mortality 0%, sympt om-free at discharge 41.5 %). After multivariate analysis, absence of disabili ty at discharge, hypertension, diabetes, hyperlipidemia, and thalamic(56.5 %) a nd corona radiata (4 %) locations were clinical and topographic variables signi ficantly associated with pure sensory stroke. Conclusions: Pure sensory stroke i s an infrequent cerebrovascular syndrome, in which the lacunar hypothesis is sup ported. Most patients had thalamic lacunar infarction. Incomplete hemisensory sy ndromes were also caused by a lacunar infarct in 84%of patients. Hemorrhagic pu re sensory stroke accounted only for 3%of the cases. The prognosis is good with striking similarity to other lacunar strokes. There are important differences b etween pure sensory stroke and nonlacunar strokes.
Objective: We report a clinical description of pure sensory stroke based on da ta collected from a prospective acute stroke registry. Methods: From 2500 acute stroke patients included in a hospital-based prospective stroke registry over a 12-year period, 99 were identified as having Pure sensory stroke was 4.7% of all acute stroke patients, 5.4% of acute is chemic stroke, and 17.4% of lacunar syndromes. Complete hemisensory syndrome was present in 80 patients and incomplete hemisensory syndrome in 19 (cheirooral syndrome 12, cheiro-oral-pedal 6, isolated oral syndrome1). The lacunar hypothes was was fulfilled in 88% of patients. Atherothrombotic infarction occurred in 8 pa tients, intracerebral hemorrhage in 3, and stroke of undetermined cause in 1. Hem orrhagic pure sensory stroke was diagnosed in 1% of all cases of hemorrhagic str oke (n = 270) in the database. Outcome was good (in-hospital mortality 0%, symptom-free at discharge 41.5%). After multivari ate analysis, absence of disabili ty at discharge, hypertension, diabetes, hyperlipidemia, and thalamic (56.5%) a nd corona radiata (4%) locations were clinical and topographic variables signi ficantly associated with pure sensory stroke. an infrequent cerebrovascular syndrome, in which the lacunar hypothesis is sup ported. Most patients had thalamic lacunar infarction. Incomplete hemisensory syndromes were also caused by a lacunar infarct in 84% of patients. Hemorrhagic pu re sensory stroke accounted only for 3% of the cases. The prognosis is good with striking similarity to other lacunar strokes. There are important differences b etween pure sensory stroke and nonlacunar strokes.