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肺本身异常限制肺泡的通气,肺周围的结构异常阻止肺的膨胀,都会引起呼吸衰竭。但是即使呼吸器官基本完好无损,而脑干的呼吸中枢或其神经元的联系障碍,呼吸作用仍可受损。本文将集中讨论肺本身基本正常而呼吸驱动的缺陷或其转化为胸廓扩张的功能不全。即可能由于呼吸中枢的调节改变,神经传导的异常,肌无力或胸壁畸形所致。一般来说上述这些情况下的临床表现取决于以下原因,例如,具有上行的神经病变时,呼吸衰竭在几小时内即能发生,病人将产生呼吸急促、气短,病人的动脉血气恶化,发绀和精
Lung itself abnormally restricting the alveolar ventilation, abnormal lung structure to prevent the expansion of the lungs, can cause respiratory failure. However, respiration can be compromised even though the respiratory organ is essentially intact and the brainstem’s respiratory center or its neurons are out of contact. This article will focus on the lung itself, the basic normal breathing-driven defects or its conversion into thoracic dilation of insufficiency. That may be due to changes in the regulation of the respiratory center, nerve conduction abnormalities, muscle weakness or chest wall deformity caused. In general, clinical manifestations in these situations are due to the following reasons. For example, respiratory failure can occur within a matter of hours with an ascending neuropathy. The patient will have shortness of breath, shortness of breath, exacerbation of the patient’s arterial bloodstream, cyanosis and fine