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铝是贫血的原因。Elliott 1978年描述了贫血和铝中毒的关系。有13例透析脑病综合征病人的透析用水中,平均铝含量明显高于40例未患该症病人透析用水的平均铝含量(P<0.01;400±33.3mg/l对<30mg/l)。回顾病程时发现,透析脑病综合征患者的Hb浓度降低先于神经系统症状出现。患者血浆铝含量非常高但无铝中毒症状。据此推测全身性不适、呕吐、体重减轻、Hb降低和肌肉或骨痛是铝引起的透析脑病先兆征象。Short等报告了12例血液透析病人发生严重小细胞低色素非缺铁性贫血,其血浆铝含量都增高。其中8例后来出现铝中毒的临床和组织学证据,铝亦来源于透析用水。后经逆渗透作用处理
Aluminum is the cause of anemia. Elliott 1978 describes the relationship between anemia and aluminum poisoning. In 13 patients with dialysis encephalopathy syndrome, the average aluminum content in dialysis water was significantly higher than that of 40 patients without the disease (P <0.01; 400 ± 33.3 mg / l vs. <30 mg / l). Review of the course of the disease found that patients with dialysis encephalopathy decreased Hb concentration before the emergence of neurological symptoms. Patients with high plasma aluminum but no symptoms of aluminum poisoning. Presumably, generalized malaise, vomiting, weight loss, decreased Hb, and muscular or bone pain were the precursors of dialysis encephalopathy caused by aluminum. Short et al. Reported the occurrence of severe microcytic hypopigmentation non-iron-deficiency anemia in 12 hemodialysis patients with elevated plasma aluminum levels. Eight of the patients subsequently developed clinical and histological evidence of aluminum toxicity and aluminum was also derived from dialysis water. After the reverse osmosis treatment