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Aims Intestinal ischemia-reperfusion (IIR) has been established as an important pathophysiological process to multiple organ dysfunctions in critical patients.Recent studies reported that dual expression programs of the B cells receptors and Toll receptors on B-lymphocytes permit these ubiquitous cells to integrate both adaptive and innate immune functions.Our previous study found that somatostatin (SST) inhibited the intestinal inflammatory injury after IIR in macaques; however, the changes of B cells and the effects of SST on B cells after IIR were unclear.