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Purpose Idiopathic pulmonary fibrosis (IPF) is defined as a form of fatal,progressively fibrosing pulmonary interstitial disease of unknown etiology, and practically no effective therapy has yet been established.Our experiment is estabilished to explore the mechanism of exogenous ACE2 ameliorating lung fibrosis at different stages of disease progression in animal model, thus providing a potential target for the therapy of IPF.