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  5. Cathepsin S activity controls injury-related vascular repair in mice via the p38MAPK and PI3K-Akt/p-

Cathepsin S activity controls injury-related vascular repair in mice via the p38MAPK and PI3K-Akt/p-

来源 :Ready for 2018- the 4th China International Forum on Hyperte | 被引量 : 0次 | 上传用户：francis123123
【摘 要】
：
Background:Neointimal hyperplasia is a major cause of restenosis after coronary intervention.The role of cathepsin S (CatS) in endovascular treatment injury-induced neointimal hyperplasia is poorly un
【作 者】
：
Hongxian Wu Xianwu Cheng Kyosuke Takeshita Lina Hu Chen Hu Qiuna Du Zhe Huang Xiaohong Zhang Masafumi Kuzuya Guoping Shi Qiuyan Dai Toyoaki Murohara 
【机 构】
：
Department of Cardiology,Nagoya University Graduate School of Medicine,Nagoya,Japan;Department of Ca
【出 处】
：
Ready for 2018- the 4th China International Forum on Hyperte
【发表日期】
：
2015年3期
【关键词】
：
cathepsin S neointimal formation HDAC6 vascular remodeling 
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　　Background:Neointimal hyperplasia is a major cause of restenosis after coronary intervention.The role of cathepsin S (CatS) in endovascular treatment injury-induced neointimal hyperplasia is poorly understood.Our findings described herein revealed that CatS has a role in injury-related vascular repair in mice.
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