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The effects of vagal and sympathetic nerves on the transmembranepotentials of cardiac cells of toad were observed by means of microelectrodetechnique.The vagal nerve was stimulated there would be an increase in restingpotential and acceleration in repolarization of action potential(AP).However,ifatropine was used before stimulation the above-mentioned phenomena woulddisappear.When the sympathetic nerve was stimulated the AP amplitudeincreased,but resting potential(RP)remained the same.The increase of APresulted from the increases of overshoot.When the sympathetic nerve wasstimulated although the heart rate increased and the duration of AP wasshortened,the plateau phase of AP was prolonged.These results suggest that theeffects of vagal and sympathetic nerves on the transmembrane potential of cardiacventricular cells are coordinated and the normal characteritics of transmembranepotential are maintained by both the vagal and sympathetic nerves.
The effects of vagal and sympathetic nerves on the transmembrane potential of catheters of toad were observed by means of microelectrodetechnique. The vagal nerve was stimulated there would be an increase in resting potential and acceleration in repolarization of action potential (AP) .However, ifatropine was used before stimulation the above-mentioned devices would like to stimulate the AP amplitude increased, but resting potential (RP) remained the same. The increase of APresulted from the increases of overshoot. When the sympathetic nerve wasstimulated although the heart rate increased and the duration of AP wasshortened, the plateau phase of AP was prolonged. these results suggest that theffects of vagal and sympathetic nerves on the transmembrane potential of cardiacventricular cells are coordinated and the normal characteritics of transmembranepotential are maintained by both the vagal and sympathetic nerves.