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严重脑损伤后的高血压特点如收缩压升高,心动过速,心排出血量增多,外周血管阻力正常或降低,循环中儿茶酚胺增多.收缩压升高可导致颅内压增高并引起脑的继发性损害.因此有人建议维持或降低收缩压在160mmHg以下以防止继发性损害,也有运用治疗原发性高血压的常用药物——血管舒张剂达到防止继发性损害的目的.然而,原发性高血压病人心排血量正常,全身血管阻力升高,与继发于脑损伤的高血压血液动力学的改变恰相反.所以,治疗原发性高血压的药物对伴有脑损伤的高血压疗效可能无效,甚至有害.
Hypertension after severe brain injury such as increased systolic blood pressure, tachycardia, increased cardiac output, peripheral vascular resistance normal or reduced circulation increased catecholamines increased systolic blood pressure can lead to increased intracranial pressure and cause brain Secondary damage.So it was suggested to maintain or reduce systolic blood pressure below 160mmHg in order to prevent secondary damage, but also the use of commonly used drugs for the treatment of essential hypertension - vasodilator to achieve the purpose of preventing secondary damage.However, Essential cardiac output in patients with essential hypertension, systemic vascular resistance increased, and secondary to cerebral injury in the opposite hemodynamic changes in hypertension, therefore, the treatment of essential hypertension drugs associated with brain injury Hypertension may be ineffective and even harmful.