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近时大量的研究资料表明,脂质过氧化作用不仅参与某些正常的生理过程,而且还与许多病理过程密切相关[见中华医学杂志1987;67(12):641]。矽肺是一种严重危害煤矿按尘工人身体健康的职业病,其基本病理变化是肺巨噬细胞吞噬粉尘损伤后所致的肺慢性进行性结节型纤维化。虽然国外已有矽肺和过氧化损伤相关研究的报道,但国内对矽肺机体内脂质过氧化作用及其生物学意义的研究却不多。本文采用
A large number of recent studies have shown that lipid peroxidation not only involved in some normal physiological processes, but also with many pathological processes are closely related [Zhonghua Medical Journal 1987; 67 (12): 641]. Silicosis is a serious endanger the health of coal mine workers by dust occupational diseases, the basic pathological changes of pulmonary macrophages phagocytosis after dust damage caused by chronic pulmonary nodular fibrosis. Although there are reports about the related research of silicosis and peroxidation in foreign countries, there is not much research on the lipid peroxidation in vivo and its biological significance in China. This article uses