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由于精神紧张或肥胖等因素造成的高血压,其患者体内分泌的去甲肾上腺素、肾上腺素等儿茶酚胺较常人更多。儿茶酚胺作用于效应受体,促使心输出量增加,外周阻力血管不断收缩,从而血压升高。并且,儿茶酚胺对高血压患者产生的效应,又较常人高,于是促使血压进一步升高。高血压又可造成心脏肥大、动脉粥样硬化或心律失常。病理原因既由于儿茶酚胺,用肾上腺素神经β受体阻断药(如普萘洛尔)或α_1受体阻断药(如哌唑嗪)治疗,自可产生有益的效应。更进一
Due to stress or obesity and other factors caused by hypertension, the patient’s body secretion of norepinephrine, adrenaline and other catecholamines more than the average person. Catecholamines act on the receptor, and promote increased cardiac output, peripheral resistance blood vessels continue to shrink, resulting in elevated blood pressure. Moreover, the effect of catecholamines on hypertensive patients, but also more common people, so to promote further increase in blood pressure. Hypertension can cause cardiac hypertrophy, atherosclerosis or arrhythmia. Pathogenesis causes both beneficial effects due to catecholamines, treatment with beta-blockers of the adrenergic neurons, such as propranolol, or alpha-1 blockers, such as prazosin. More into one