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缺氧对心脏功能的影响与缺氧的严重程度、发生速度及时程有关。本实检比较了急性缺氧与阶梯适应性缺氧对Ｗｉｓｔａｒ大鼠心脏功能及心肌收缩蛋白Ｃａ２＋，Ｍｇ２＋－ＡＴＰ酶的不同影响，结果表明，低氧适应组与急性缺氧组比较，左右心室的±ｄｐ／ｄｔｍａｘ、收缩指数等心功能指标均有显著的改善，心肌收缩蛋白Ｃａ２＋，Ｍｇ２＋－ＡＴＰ酶活性也显著高于急性缺氧组。从而说明，动物经低氧适应后，心脏的代偿功能得到充分发挥，从面减轻缺氧对心脏的损伤。心肌收缩蛋白Ｃａ２＋，Ｍｇ２＋－ＡＴＰ酶的改善可能是心脏代偿机制的生物化学基础之一。
The effect of hypoxia on cardiac function is related to the severity of hypoxia, the rate of occurrence and the time course. The actual seizure compared acute hypoxia and step adaptation hypoxia on Wistar rats cardiac function and cardiac contractile protein Ca2 +, Mg2 + -ATP enzyme of different effects, hypoxia adaptation group and acute hypoxia group, left and right ventricular ± dp / dtmax, systolic index and other cardiac function indicators were significantly improved myocardial contractile protein Ca2 +, Mg2 + -ATPase activity was significantly higher than the acute hypoxia group. Thus, animal hypoxia adaptation, the compensatory function of the heart has been fully exerted, from the surface to reduce hypoxic heart damage. The improvement of myocardial contractile proteins Ca2 +, Mg2 + -ATPase may be one of the biochemical basis of cardiac compensatory mechanism.