论文部分内容阅读
牙周炎的发病机制,包括细胞免疫功能受抑制,白细胞趋化性和吞噬功能降低。作者研究牙周炎患者白细胞吞噬功能,以及用淋巴因子矫正吞噬功能障碍的可能性,检查中型和重型牙周炎10例(基本组),无伴发病,年龄22岁～56岁。检查无牙周病的健康志愿者5例(对组照)。从指尖和龈各取血0.1ml,微量法用胶乳测定中性白细胞吞噬功能。为研究淋巴因子对白细胞吞噬功能的调节,从基本组、对照组和健康供血者的静脉抽血,分离出淋巴细胞进行培养,加入植物血凝素刺
The pathogenesis of periodontitis, including inhibition of cellular immune function, leukocyte chemotaxis and phagocytic function decreased. The authors studied leukocyte phagocytosis in patients with periodontitis, and the possibility of correcting phagocytic dysfunction with lymphokines. Ten patients (median group) with moderate and severe periodontitis were examined for absence of disease, ranging in age from 22 to 56 years. Five healthy volunteers who had no periodontal disease were examined (pairwise). From the fingertips and gingival bleeding 0.1ml, trace method with latex neutrophil phagocytosis. In order to study the regulation of leukocyte phagocytosis by lymphokines, blood was drawn from the venous blood of basic group, control group and healthy donors, lymphocytes were isolated and cultured, and then phytohemagglutinin