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作者报告了一例误服氰化物中毒造成肌张力障碍和提睑不能的帕金森综合征,并进行了CT扫描和MRI研究,用抗胆硷药和三已芬迪,金刚胺等治疗有一定缓解.46岁女性患者,与丈夫一起饮用了一瓶掺杂氰化物的软饮料后昏迷,其丈夫送医院途中死亡.该患者入院时浅昏迷,右侧Babinski征阳性,CT和脑脊液检查均正常、脑电图有弥漫慢波活动,胃液分析证实氰化物中毒.不久病人呈去脑强直状态,2周后逐渐好转,出现发音困难和构音障碍.右侧肢体轻瘫,腱反射亢进、Babinski征阳性.一年后病人因言语增多和吞咽困难、流口水和步态不稳而转院,CT扫描示中度皮层萎缩.脑电图示颞叶导程有散在的非特异性改变.神经心理检查显示对空间和视觉记忆障碍,对视知觉操作和抽象理解差,继而
The authors report a case of Parkinsonism caused by cyanosis poisoning that causes dystonia and eyelid inability to do so and performed CT scan and MRI studies with some relief with anticholinergic agents such as anticonvulsant and triamcinolone A 46-year-old woman, co-drinking a bottle of cyanide-laden soft drink with her husband, left her husband on her way to hospital with a coma, positive Babinski sign, CT and cerebrospinal fluid were normal, brain Electromyography diffuse slow wave activity, gastric juice analysis confirmed cyanide poisoning. Shortly after the patient showed an ankylosis, gradually improved after 2 weeks, there is dysarthria and dysarthria. Right limb paresis, tendon hyperreflexia, Babinski sign positive One year later, the patient was transferred to hospital due to increased speech and difficulty swallowing, drooling, and unsteady gait, and CT scans showed moderate cortical atrophy. EEG showed scattered nonspecific changes in the temporal lobe lead. Spatial and visual memory impairment, poor perception of visual perception and abstract understanding, and then