论文部分内容阅读
《近年来,由于加压(正压)给氧受到重视和改进,使多数所谓“难治性肺水肿”也获得较好疗效。正压呼吸不但可提高通气量,也有利于气体在肺脏的均匀分布。在压力较大、持续时间充裕的条件下,气体可进入弹力减退或气道阻力增加的肺泡,从而减少生理无效腔和生理性动静脉分流。然而,正压呼吸对心脏循环有不利的一面,能使血压下降和使肺循环阻力增加,加重右心负担。因此,正确使用正压呼吸需要选用恰当的压力、呼吸时间和呼吸比率,既要保证有效的通气量和气体交换,又要使心脏循环的负担减至最小限度。此文通过对24篇文献的复习,就正压终末呼气压对心脏的影响机理进行探讨——译注》
In recent years, due to the pressure (positive pressure) oxygen has been valued and improved, so that most so-called \"refractory pulmonary edema,\" also received better results. Positive pressure breathing can not only improve ventilation, but also conducive to the uniform distribution of gas in the lungs. Under stressful conditions and prolonged periods of time, the gas can enter the alveoli with diminished elasticity or increased airway resistance, thereby reducing the physiologic void and physiologic arteriovenous shunts. However, positive pressure breathing has a negative side of the heart circulation, can make blood pressure and pulmonary circulation resistance increased, increasing the burden on the right heart. Therefore, the correct use of positive pressure breathing need to choose the right pressure, breathing time and respiratory rate, both to ensure effective ventilation and gas exchange, but also to minimize the burden of heart circulation. Through the review of 24 articles, this paper discusses the mechanism of the effect of end-pressure of end-pressure on the heart.