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已知妊娠诱发的高血压(PIH)是胎儿与母体死亡的主要原因,据报道初孕妇中发病率高达10％。PIH的病理生理机制尚未明确,一个广为接受的事实是,产生于正常妊娠中的周围血管阻力降低和动脉血压降低现象,在PIH中缺如或相反增加。最近一些初步报道提示:使用显微神经学的技术测定,发现PIH及先兆子痫比正常妊娠中交感神经的活动更多。而所有这些报告中,多单元肌肉交感神经活动(MSNA)是用处理过的猝发冲动(Burst)来测定的。该文的目的,是通过直
Pregnancy-induced hypertension (PIH) is known to be the leading cause of fetal and maternal deaths and is reported to occur in up to 10% of first-time mothers. The pathophysiologic mechanism of PIH is unclear. It is a widely accepted fact that the reduction of peripheral vascular resistance and arterial blood pressure arising from normal pregnancy is absent or conversely increased in PIH. Some recent initial reports suggest that using microneurological techniques, PIH and preeclampsia were found to have more sympathetic activity than normal pregnancies. In all of these reports, multiple unit muscle sympathetic nerve activity (MSNA) was measured using treated burst. The purpose of this article is through Straight