论文部分内容阅读
患者男,70岁,以脑梗死于1999年7月入院。就诊时呈浅昏迷,双侧巴氏征(+)。输入葡萄糖液后意识恢复正常,经颅脑CT检查示腔隙性脑梗死。体格检查:体温、呼吸、脉搏和血压均正常,意识清,语言清,颅神经正常,心、肺、肝脾无异常,四肢肌力正常,无病理反射。入院后给予706代血浆、胞二磷胆碱、脉络宁等药物治疗,治疗过程中再次出现意识障碍并双侧巴氏征(+)。速查血糖2.4mmol/L,即刻给予50%葡萄糖40ml静脉注射,病情恢复正常,病理反射消失。此后反复发作数次,血糖均低,均经同样处置后清醒。进一步做肝脏彩超及CT,诊断为肝右叶后段肝癌,甲胎蛋白
Male patient, 70 years old, was admitted to hospital in July 1999 with cerebral infarction. Was shallow coma at the time of treatment, bilateral Pakistan’s sign (+). After the glucose solution was returned to normal consciousness, the brain CT examination showed lacunar infarction. Physical examination: body temperature, breathing, pulse and blood pressure were normal, clear consciousness, clear language, normal cranial nerves, heart, lung, liver and spleen were normal, normal muscle strength, no pathological reflex. After admission to give 706 generations of plasma, citicoline, Mailuoning and other drug treatment, again in the process of disturbance of consciousness and bilateral Pakistan’s sign (+). Quickly check the blood glucose 2.4mmol / L, immediately give 50% glucose 40ml intravenous injection, the condition returned to normal, pathological reflex disappeared. After repeated seizures several times, blood sugar are low, all by the same treatment after awake. Further liver and color Doppler ultrasound CT, the diagnosis of liver cancer after the right lobe, a fetoprotein