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1.低浓度阿司匹林(80～160毫克/日)已可使80%血小板膜的环氧化酶被抑制,血小板产生血栓烷A_2(TxA_2)减少,血小板聚集受抑制,出血时间延长但小剂量阿司匹林不会抑制血管壁的前列环素(PGI_2)合成酶,PGI_2的生成不受限制,后者为抗血小板聚集剂。常规剂量阿司匹林(0.3克每天3次)或大剂量阿司
1. Low concentrations of aspirin (80 ~ 160 mg / day) has been 80% of the platelet membrane cyclooxygenase is inhibited, platelets produce thromboxane A_2 (TxA_2) reduction, inhibition of platelet aggregation, prolonged bleeding but low-dose aspirin Does not inhibit the prostacyclin (PGI_2) synthase on the vascular wall, and the production of PGI_2 is not limited, and the latter is an anti-platelet aggregation agent. Regular doses of aspirin (0.3 g 3 times daily) or high-dose asparagus