论文部分内容阅读
目的通过使用阿托品对36例急性颅脑损伤患者引起的心动过缓的治疗效果进行分析。结果治疗后1 h心率>90次/min 12例;60次/min<心率<90次/min 13例;50次/min<心率<60次/min 7例;40次/min<心率<50次/min 4例;复查心电图均未发现改变。入院后第7天复查心肌酶学:异常5例,所有患者均未出现心慌、胸闷等症状,全部停止使用阿托品,其心率均未见明显改变,亦未再次出现心慌、胸闷等症状。入院第14 d复查心肌酶学,均未异常,36例中均无心慌、胸闷等症状。其中心率<60次/min 8例,心率<50次/min 2例,出院后一个月复查36例心率均>60次/min,亦无心慌、胸闷等症状。结论阿托品治疗急性颅脑损伤引起的心动过缓有效,但对急性颅脑损伤引起的心动过缓、心肌酶学异常而无心慌、胸闷等症状者效果欠佳,其个别不需要治疗,即使心肌酶学异常,心率小于50次/min亦无需特殊处理而自然好转。
Objective To analyze the therapeutic effect of atropine on bradycardia in 36 patients with acute craniocerebral injury. Results In 1 hour after treatment, heart rate> 90 beats / min in 12 cases, 60 beats / min 60 beats / min after discharge one month after discharge. There were no symptoms such as palpitation and chest tightness. Conclusion Atropine treatment of acute traumatic brain injury caused by bradycardia effective, but acute bradycardia caused by bradycardia, myocardial enzyme abnormalities without palpitation, chest tightness and other symptoms were ineffective, and its individual does not require treatment, even if the myocardium Enzyme abnormalities, heart rate less than 50 times / min and no special treatment and natural improvement.