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本文报告2例皮肤粘膜淋巴结综合征的临床表现,讨论了病因可能与细菌、病毒、原虫、支原体等多种病原体有关。病例1血培养为葡萄球菌,病例2血沉、抗“O”增高与链球菌感染有关。其机理可能是早期给药细菌被破坏,由菌体成份引起机体赫氏样反应。有的病例发病前有前驱感染,免疫复合物及免疫球蛋白升高,表明很可能为感染原刺激机体后产生异常应答反应,导致自身免疫过程,是以血管为中心的过敏性病变。
This article reports 2 cases of clinical manifestations of mucocutaneous lymph node syndrome, discussed the etiology may be related to bacteria, viruses, protozoa, mycoplasma and other pathogens. Case 1 blood culture for Staphylococcus, Case 2 ESR, anti-“O” increased and streptococcal infection. The mechanism may be early administration of bacteria is destroyed, the body composition caused by the body Hercules-like response. In some cases before the onset of a precursor infection, immune complexes and immunoglobulin increased, indicating that it is likely to produce abnormal response to the body after the original infection response, leading to autoimmune processes, is a vascular-centered allergic disease.