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6例病人中,1例分娩时出现DIC,另5例首次检查并未提示任何止血功能缺陷,以后发现纤维蛋白原/纤维蛋白降解产物增加,血小板减少。5例血浆鱼精蛋白副凝试验阳性,提示循环纤维蛋白单体存在。例2有自发的血小板凝集,可被肝素抑制。3例有明显的低纤维蛋白原血症,或不相称的纤维蛋白原降低,2例凝血酶原时间稍延长,某种凝血因子降低。4例末梢血片有裂细胞存在。 6例中无1例有明显诱发DIC的原因。5例进一步肺扫描证实有肺性塞,1例由血管造影证实。在
Of the 6 patients, 1 developed DIC at delivery and the other 5 did not show any hemostatic defects for the first time, and subsequently increased fibrinogen / fibrin degradation products and thrombocytopenia were found. Five cases of plasma protamine paraidineide test positive, suggesting the presence of circulating fibrin monomer. Example 2 has spontaneous platelet aggregation, can be inhibited by heparin. Three cases had marked hypofibrinogenemia, or disproportionate reduction of fibrinogen, two cases of prothrombin time slightly longer, some kind of clotting factor decreased. 4 cases of peripheral blood cells have split cells. None of the 6 patients had a clear cause of DIC. Pulmonary plugs were confirmed in 5 cases with further lung scans and in 1 case confirmed by angiography. in