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急性心肌梗塞合并WPW早已为临床所重视,而合并LGL尚少有报道。我院曾见一例,报告如下。施某,女性,67岁(住院号79—3400)。因五天来胸骨后疼痛进行性加剧而于1979年5月11日来院急诊。发病初疼痛尚轻呈阵发性,三天后疼痛加剧,且持续时间延长,并向左肩放射,伴胸闷、气急,因疼痛持续一小时不缓解而急诊住院。既往有高血压病史十余年,两年前曾进行心电图描记未发现异常。体检:T.35℃,P 65次/分,B P 56/40mmHg,神志清楚,急性病容,肥胖体型,口唇稍紫,颈软,颈静脉不怒张。心界向左扩大,心律齐,第一心音减低,A_2>P_2,两肺呼吸音无异常。肝脾无明显肿大,下肢无浮肿。入院后当即描记心电图示房室交界性心律,电轴不偏。次日复查结果为窦性心律,LGL综合征,急性下壁心肌梗塞(与前日比较V_(1-4)之ST段抬高略呈弓背向上)。住院期间经吸氧、低分子右旋糖酐、丹参、极化液及抗休克治疗后患者血压回升、尿量
Acute myocardial infarction with WPW has long been clinically important, and the merger of LGL has yet to be reported. I have seen a hospital, the report is as follows. Shi, female, 67 years old (hospital number 79-3400). Hospitalized for emergency on May 11, 1979, due to the progressive increase of post-sternal pain for five days. The incidence of early pain was mild paroxysmal, three days after the pain intensified, and the duration of the extension, and to the left shoulder radiation, with chest tightness, shortness of breath, pain for one hour without remission and emergency hospitalization. Past history of hypertension more than ten years, two years ago had ECG was not found abnormalities. Physical examination: T.35 ℃, P 65 beats / min, B P 56 / 40mmHg, conscious, acute disease, obese body shape, a little purple lips, neck soft, jugular vein does not anger. Heart to the left to expand, rhythm Qi, the first heart sound reduced, A_2> P_2, both lung breath sounds no abnormalities. Liver and spleen no obvious enlargement, lower extremity no edema. ECG immediately after admission, atrioventricular junctional arrhythmias, electrical axis is not partial. The next day’s results were sinus rhythm, LGL syndrome, acute inferior myocardial infarction (compared with the previous day V_ (1-4) ST segment elevation slightly bow back up). During hospitalization by oxygen, low molecular weight dextran, Salvia, polarized fluid and anti-shock treatment in patients with blood pressure rise, urine output