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近10年来由于CT扫描技术的发展使腔隙性梗塞得以作出临床诊断。现将有关本病近年来某些研究进展综述如下。一、病因及病理高血压一直被认为是本病的直接病因,尤其是慢性高血压超过160/95对。持续性高血压作用于小动脉及微血管壁,引起管壁的节段性脂肪透明变性、类纤维蛋白坏死及微动脉粥佯瘤等改变,导致小动脉阻塞而形成梗塞;然后由大吞噬细胞清除梗塞的脑组织,留下的腔或小孔即所谓的腔隙。但临床上有25～53%
In the past 10 years due to the development of CT scanning technology to make lacunar infarction to make a clinical diagnosis. Now the disease in recent years, some research advances are summarized below. First, the etiology and pathology Hypertension has been considered the direct cause of the disease, especially over 160/95 of chronic hypertension. Persistent high blood pressure in the arterioles and microvascular wall, causing the wall of the segmental fat transparent degeneration, fibrinogen necrosis and micro-arterial tumor xenografts and other changes, leading to obstruction of the arterioles and the formation of infarction; and then by the macrophage clearance Infarcted brain tissue, leaving cavities or small holes called cavities. However, there are clinically 25 to 53%