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本文旨在对急性心肌梗死(AMI)后卡托普利治疗的即期血流动力学、神经内分泌生化学,及对远期临床症状、左室结构和运动耐量的可能影响进行了研究。病人与方法 99例临床症状及血流动力学稳定的AMI患者(男82例,女17例,年龄40～75岁),于胸痛24小时内纳入研究。先期排除小范围心梗、心室扩张低危者。并除外SBP<95mmHg(12.7kPa,1kPa=7.5mmHg)或患有严重脑、肾血
This article aims to investigate the immediate hemodynamics, neuroendocrine biochemistry, and possible effects on long-term clinical symptoms, left ventricular structure, and exercise tolerance after acute myocardial infarction (AMI) with captopril. Patients and Methods Ninety-nine patients with clinical symptoms and hemodynamic-stable AMI (82 males and 17 females, aged 40-75 years) were included in the study within 24 hours of chest pain. Early rule out a small area of myocardial infarction, low ventricular dilatation. With SBP <95 mmHg (12.7 kPa, 1 kPa = 7.5 mmHg) or with severe brain, kidney blood