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本文对60例高血压经股动脉穿刺冠状动脉造影患者随机分组为高血压组20例,不干预血压;硝苯定组40例,采用硝苯定舌下含服降血压,以观察对股动脉穿刺部位出血止血时间及血肿的作用,并以40例血压正常的患者作为对照组。结果显示:高血压组与血压正常组、硝苯定组相比出血止血时间明显延长,(40.70±6.53min 对20.75±2.44min,25.10±3.95min,分别 P<0.001,P<0.05)差别有显著性,而血压正常组和硝苯定组之间差别无显著性;高血压组局部大(>3cm)、中(1～3cm)血肿明显高于血压正常组及硝苯定组,差别有显著性,血压正常组和硝苯定组之间差别无显著性提示:硝苯啶疔防止高血压所致股动脉穿刺部位出血止血时间延长及减少血肿发生。
In this paper, 60 cases of hypertension by femoral artery puncture coronary angiography were randomly divided into hypertension group of 20 patients, do not interfere with blood pressure; nifedipine group of 40 patients with nifedipine sublingual hypotension to observe the femoral artery Puncture site hemostasis bleeding time and hematoma role, and to 40 cases of normotensive patients as a control group. The results showed that the hemostasis time of hypertensive patients was significantly longer than that of nifedipine and nifedipine groups (40.70 ± 6.53min vs. 20.75 ± 2.44min, 25.10 ± 3.95min, respectively, P <0.001, P <0.05) (N = 3), moderate (1-3 cm) hematoma in hypertensive group was significantly higher than that in normotensive and nifedipine groups, the difference was significant No significant difference between the normotensive and nifedipine groups was observed. Nifedipine prevented the hemostasis time of hemorrhage caused by hypertensive femoral artery punctures and reduced the occurrence of hematoma.