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稻瘟病菌９０－２菌株系是从大田水稻雄性不育细胞质（ＣＭＳ）上分离的对野败型（Ｗ）ＣＭＳ专化致病的生理小种。研究表明，该菌株产生的毒素在致病过程中起着一定作用。本试验测定了此菌株在不同ｐＨ培养基中培养对毒素产生的影响、毒素的热稳定性及ｐＨ值变化对毒素活性的影响等。离体叶片法测定毒素活性发现毒素使离体叶段褪绿和细胞坏死，珍汕９７Ａ褪绿程度大于珍汕９７Ｂ，进一步测定了毒素对Ｗ珍汕９７Ａ、９７Ｂ叶绿素总含量的影响和对Ｗ珍汕９７Ａ、９７Ｂ呼吸作用的影响，初步测定了粗毒素的致病作用。用根冠细胞法测定了９０－２菌株毒素对一套４种同核异质ＣＭＳ材料的致病力，表明稻瘟病菌９０－２菌株是ＣＭＳ野败型的专化生理小种。
The strain of Magnaporthe grisea 90-2 was isolated from male sterile cytoplasm (CMS) of Oryza sativa. Studies have shown that toxins produced by this strain play a role in the pathogenesis. In this experiment, we measured the effect of the strain on the toxin production, the heat stability of the toxin and the change of the pH on the toxin activity in different pH media. In vitro leaf method was used to determine the toxin activity. It was found that the toxin caused the chlorotic and necrotic cells in isolated leaf segments. The degree of chlorosis of Zhenshan 97A was greater than that of Zhenshan 97B, and the effects of toxins on the total content of chlorophyll in Wanshan 97A and 97B were further determined. The effects of respiration of Zhenshan 97A and 97B were studied. The pathogenic effects of crude toxin were preliminarily determined. The pathogenicity of a strain of 90-2 strain to a set of four isonuclear allogeneic CMS materials was determined by root-crown cell assay, indicating that M. grisea 90-2 strain is a specialized race of CMS wild-type.