论文部分内容阅读
低血磷可由多种病理情况产生。Fuller等曾注意到低血磷,特别是伴有严重肌负荷可促发横纹肌分解。本文病人由于甲状旁腺切除术及补钙继发严重的低血磷,一度发生横纹肌分解。为控制肾性骨营养不良症,在进行长期透析治疗的病人常需作甲状旁腺切除术。据作者所知,以往没有报道过横纹肌分解作为甲状旁腺切除术及补钙的并发症。一个长期血透的33岁女病人,1959年曾诊为基膜性肾小球肾炎,1973年起作长期透析治疗,病程中并发严重高血压,于1973年10月作双侧肾切除。在血透期间血清无机磷水平保持在2.8～3.4mg/dl,无需服用磷结合剂,但发生了肾性骨营养不良症,X线表现为:肋骨骨折及骨痛等。甲旁激素水平为19.6ng/ml(正常0～1.5ng/ml)。1977年3月29日作甲状旁腺
Hypophosphatemia can be produced by a variety of pathological conditions. Fuller et al have noted hypophosphatemia, especially with severe muscle load can trigger striated muscle decomposition. This patient due to parathyroidectomy and calcium secondary to severe hypophosphatemia, once the occurrence of striated muscle decomposition. In order to control renal osteodystrophy, patients undergoing long-term dialysis often require parathyroidectomy. To the authors’ knowledge, there have been no reports of rhabdomyolysis as a complication of parathyroidectomy and calcium supplementation in the past. A long-term hemodialysis of 33-year-old female patient, 1959 was diagnosed with basal membranous glomerulonephritis, long-term dialysis in 1973 for treatment, the course of severe concurrent hypertension, in October 1973 for bilateral nephrectomy. During hemodialysis serum inorganic phosphorus levels remained at 2.8 ~ 3.4mg / dl, without taking phosphate binders, but the occurrence of renal osteodystrophy, X ray showed: rib fractures and bone pain and so on. Parathyroid hormone levels of 19.6ng / ml (normal 0 ~ 1.5ng / ml). March 29, 1977 for the parathyroid