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多发硬化（MutipleSclerosisMS）目前被认为是T细胞介导的中枢神经系统慢性自身免疫脱髓鞘疾病，病程为缓解复发，慢性进行性加重，终至功能丧失。在病变发展过程中．细胞因子发挥了重要作用。肿瘤坏死因于一a（TNF－a）、白介素一6（11。－6）等均可促进此免疫反应导致脱髓鞘?
Mutiple Sclerosis MS is currently considered as a T-cell-mediated central nervous system chronic autoimmune demyelinating disease with a course of relapse, chronic progressive exacerbation, and finally loss of function. During the development of the lesion. Cytokines play an important role. Tumor necrosis due to a (TNF-a), interleukin-6 (11.-6), etc. can promote this immune response leads to demyelination?