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既往多认为肢体失用症是由于优势半球的额顶部皮质和/或与之发生联系的同侧半球纤维束和通过胼胝体与之发生联系的对侧运动前区皮质损害而引起。但作者注意到一例经CT 证实为左侧丘脑、内囊后肢和豆状核后部的血肿,出现严重的肢体失用症。本组共14例,4例为血肿,10例梗塞,均位于左侧三脑室壁与外囊之间区域。于发病后10～50天患者意识清楚且在合作的情况下进行肢体运用功能检查。包括:模仿运动试验:嘱患者用病灶对侧手重复检查者所作的24次运动,第一次或第二、三次做
Previously, limb apraxia was thought to be caused by the frontal cortex of the dominant hemisphere and / or the ipsilateral hemispherical fiber bundles associated therewith and cortical damage to the contralateral pre-motor area associated with the corpus callosum. However, the author noticed a CT-confirmed hematoma in the left thalamus, the posterior limb of the internal capsule, and the posterior portion of the lentiform nucleus, with severe limb failure. The group of 14 cases, 4 cases of hematoma, 10 cases of infarction, are located in the left ventricle between the wall and the outer capsule area. 10 to 50 days after onset of patients with clear awareness and in the case of cooperation in limb function tests. Including: imitation of exercise test: Zhu Huanzhe side of the hand repeat examination of the person who made 24 movements, the first or second, three times to do