Lipid mediators of inflammation in neurological injury: shifting the balance toward resolution

• 作 者: Jordan L.Harrison Rachel K.Rowe Jonathan Lifshitz
• 机 构: BARROW Neurological Institute at Phoenix Children’s Hospital,Interdisciplinary Graduate Program in N
• 出 处: Neural Regeneration Research
• 发表日期: 2016年01期
• 关键词: inflammation neurological homeostasis initiate protective removing vital toward

Acquired neurological injuries initiate a pathological cascade of secondary injury processes,including inflammation,which continue for days to weeks following injury.Injury-induced neuroinflammation acts as a host defense mechanism contributing to the neutralization of the insult(removing offending factors)and restoring structure and function of the brain(establish homeostasis).The timing of these protective functions of the immune response is vital,since chronic inflammation

Acquired neurological cover a pathological cascade of secondary injury processes, including inflammation, which continue for days to weeks following injury. Injury-induced neuroinflammation acts as a host defense mechanism contributing to the neutralization of the insult (removing offending factors) and restoring structure and function of the brain (establish homeostasis). The timing of these protective functions of the immune response is vital, since chronic inflammation