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作者在三年内对59例(61耳)突聋患者用血管扩张剂、类固醇、血液稀释及高压氧等进行了治疗。治疗前、后行听力学检查——包括纯音测听、语言测听、耳蜗电图描记和脑干听反应测听(ABR),后者用短声刺激并记录其阈值。治疗前先测定N_1及波V的潜伏期。结果:波V-N_1之间潜伏期<4.5ms者23例,病变在耳蜗,其中17例听力恢复满意,6例听力无增进或变坏,这些病例有的N_1的潜伏期延长或就诊过晚。蜗后病变者19例,波V-N_1间的潜伏期大于4.5m s者9例,治疗后听力亦有增进,特别是有4例用CT检查证实为听神经瘤,经药物治疗后,3例听力几乎恢复到正常水平,但脑干电反应的潜伏期仍然延长,其听力之所以能改进,可能与内耳道内被压迫血管解压有关。
In three years, 59 patients (61 ears) with deafness were treated with vasodilators, steroids, hemodilution and hyperbaric oxygen. Hearing tests, including pure tone audiometry, audiometry, electrocautery electrogram and brainstem response audiometry (ABR), were performed before and after treatment, and the latter was stimulated with short sound and its threshold recorded. Before treatment, determine the latency of N_1 and V. Results: There were 23 cases of latent period between V-N_1 <4.5ms. The lesions were in the cochlea. Among them, 17 cases had satisfactory hearing recovery and 6 cases had no improvement or deterioration of hearing. In these cases, the latency of N_1 was prolonged or was too late. 19 cases of post-cochlear lesions, wave V-N_1 latency of more than 4.5ms in 9 cases, hearing after treatment also improved, especially in 4 cases confirmed by CT examination of acoustic neuroma, after drug treatment, 3 cases of hearing almost Returned to normal levels, but the latency of brainstem response is still prolonged, the reason why the hearing can improve, may be related to the decompression of blood vessels in the inner ear canal.