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老年黄斑部变性(Senile Macular Degen-eration,SMD)是老年人致盲原因之一,目前对其发病机理尚未完全弄清。Gass 和Chandra等人认为SMD 的发病机理可能是由于脉络膜毛细血管之间基质钙化变性而增厚,以及脉络膜基底膜(Bruch 膜)增厚影响到脉络膜毛细血管床所致。Delaney 等人认为SMD病因可能与老化过程有关。而另外有些人则认为SMD 与遗传和营养不良等疾病有关,不仅仅是老年变性或老化。解剖和生理的研究显示SMD 的改变为视网膜色素上皮和脉络膜的广泛硬化,而某些微量元素则参与了视网膜色素上皮及脉络膜代谢。我们对SMD 患者血清微量元素进行了测定以寻求二者之间的关系。现将结果报导如下。对象和方法一对象:以1986年8月第二届全国眼底
Senile Macular Degeneration (SMD) is one of the causes of blindness in the elderly, and its pathogenesis is not yet fully understood. Gass and Chandra, who believe that the pathogenesis of SMD may be due to choroidal capillaries between the matrix calcified degeneration and thickening, and choroidal basement membrane (Bruch membrane) thickening affect the choriocapillaris bed caused. Delaney et al believe that the cause of SMD may be related to the aging process. Others think that SMD is linked to diseases such as heredity and malnutrition, not just senile degeneration or aging. Anatomical and physiological studies have shown that changes in SMD are extensive sclerosis of the retinal pigment epithelium and choroid while some of the trace elements are involved in retinal pigment epithelium and choroidal metabolism. We measured SMD in patients with serum trace elements to find the relationship between the two. The results are reported below. Objects and methods of an object: In 1986 August the second national fundus