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患者女,24岁,主因进行性头晕,乏力三个月,加重十余天,黑便十余次,每次量约250至300毫升,于1990年8月4日收内科治疗,既往体健,无肝炎和溃疡病史。查体:发育正常,营养中等。神清,贫血貌,周身淋巴结不肿大,心肺正常,肝脾末触及。实验室检查,血红蛋白48g/L,红细胞1.75×10~(12)/L,白细胞5.4×10~9/L,出凝血时间正常,肝功能正常。骨髓穿刺涂片报告:增生性贫血。上消化道钡餐检查来见异常。经内科保守治疗十七天不见好转转外科治疗,保守治疗期间血色素曾一度上升,后又突然下降。后行剖腹探查,发现距屈氏韧带45cm处空肠壁有一4×4cm肿物,后行区段性切除。术后病理报告为
Female patient, 24 years old, mainly due to progressive dizziness, fatigue for three months, aggravated for more than ten days, mellow ten times, each time about 250 to 300 milliliters, received medical treatment on August 4, 1990, past physical fitness No history of hepatitis and ulcers. Physical examination: Normal development and moderate nutrition. God clear, anemic appearance, lymph nodes are not swollen, normal heart and lung, liver and spleen touch. Laboratory tests showed that hemoglobin 48 g/L, red blood cells 1.75×10 12/L, leukocytes 5.4×10 9/L, normal coagulation time, and normal liver function. Bone marrow puncture smear report: hyperplastic anemia. Upper gastrointestinal barium meal inspection to see abnormal. Seventeen days after conservative treatment of internal medicine, no good transfer of surgical treatment was found. During the conservative treatment period, hemoglobin once rose, and then suddenly dropped. After laparotomy, it was found that there was a 4×4cm tumor in the jejunal wall 45cm away from the flexor ligament, followed by a partial resection. Postoperative pathology report was