论文部分内容阅读
青光眼患者视功能损害系由眼压长期升高引起。眼压高可使眼血液动力学障碍,以及眼内氧化过程减弱。眼缺氧导致视网膜等眼组织受损。作者采用可以改善眼内氧化过程的药物—5%单糖羟酪酸钠(sodium oxybutyrate)及活性抗次黄嘌呤()观察对青光眼患者视功能和血液动力学状态的影响。口服,每日3次,30天为一疗程。39例青光眼患者(男23,女16),年龄多数为50～65岁。开角型青光眼33例,闭角型3例,单眼青光眼3例。所有患者均行缩瞳治疗。其中18例由于眼压较高,曾早期手术。开始治疗时24例眼压在24mmHg以下,15例在22～31mmHg之间。详细
Visual impairment in patients with glaucoma caused by the long-term rise in intraocular pressure. High intraocular pressure can hemoptysis ocular hemodynamics, as well as intraocular oxidation process weakened. Ocular hypoxia causes damage to the retina and other ocular tissues. The authors examined the effects of 5% monosaccharide sodium oxybutyrate and active hypoxanthine on visual function and hemodynamic status in patients with glaucoma, using a drug that improves intraocular oxidation. Oral, 3 times a day, 30 days for a course of treatment. 39 patients with glaucoma (male 23, female 16), the majority of the age of 50 to 65 years old. Open-angle glaucoma in 33 cases, angle-closure in 3 cases, monocular glaucoma in 3 cases. All patients underwent miosis treatment. 18 cases of which due to high intraocular pressure, had early surgery. At the beginning of treatment, 24 patients had intraocular pressure below 24 mmHg and 15 patients between 22 and 31 mmHg. detailed