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基于胶束与球状蛋白的相似性，用胶束模拟超氧化物歧化酶（ＳＯＤ）的微环境，以促进ＳＯＤ模拟物─水杨酸铜络合物对Ｏ歧化的催化作用，达到清除Ｏ＿２的目的。本文首次用培养的巨噬细胞受刺激产生的“呼吸暴发”为Ｏ的发生源，研究了在ＣＴＡＢ、ＳＤＳ、ＴｒｉｔｏｎＸ－１００及Ｔｗｅｅｎ２０胶束体系中水杨酸铜络合物对Ｏ的清除作用。研究结果表明，以水杨酸铜－ＳＤＳ胶束体系对Ｏ的清除率最高，达９０．４％。其他胶束体系对Ｏ的歧化亦均有促进作用。
Based on the similarity between micelles and globular proteins, micelles were used to simulate the microenvironment of superoxide dismutase (SOD) to promote the catalysis of SOD mimetic-copper salicylate complex on O-disproportionation, purpose. In this paper, the first time that the “respiratory burst” stimulated by cultured macrophages was the source of O, the scavenging effect of copper salicylate complexes on O in CTAB, SDS, Triton X-100 and Tween 20 micellar systems . The results show that the salicylic acid-SDS micellar system of O highest scavenging rate of 90.4%. Other micellar systems also promote the disproportionation of O.