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血脑屏障通透性增高是造成血管源性脑水肿和炎症反应等病理学变化的主要环节。近年来,许多研究表明,在脑缺血再灌注过程中有多种介质参与了血脑屏障结构和功能的改变和破坏。
Increased blood-brain barrier permeability is the main part of the pathological changes that cause vasogenic brain edema and inflammatory response. In recent years, many studies have shown that there are many mediators involved in the structure and function of blood-brain barrier in the process of cerebral ischemia-reperfusion change and destruction.