论文部分内容阅读
子痫前期为胎盘源性妊娠期特有疾病,发生在妊娠中晚期、分娩期及产后早期,表现为高血压、蛋白尿及其他全身系统性紊乱,可伴有胎儿综合征(胎儿生长受限、宫内窘迫、围产儿病率和病死率增高等),是导致孕产妇和围产儿死亡的主要原因之一,其发病率约为5%[1]。氧化应激反应是子痫前期由基本病理生理变化阶段发展至器官损害阶段的重要联系纽带,抗氧化剂可有效减轻氧化应激。维生素C、维生素E、番茄红素等的研究显示,传统抗氧化剂对子痫前期具有预防作用,但是近年的大样本随机实验证实传统抗氧化剂无法有效预防子痫前期疾病,且可能增加胎儿的并发症。本文结合子痫前期氧化
Preeclampsia is a placental-specific pregnancy-specific disease that occurs in the late third trimester of pregnancy, childbirth and the early postpartum period, manifested as hypertension, proteinuria and other systemic systemic disorders may be associated with fetal syndrome (fetal growth restriction, Intrauterine distress, perinatal morbidity and mortality increased) is one of the major causes of maternal and perinatal deaths, the incidence rate of about 5% [1]. Oxidative stress is an important link between pre-eclampsia from basic pathophysiological changes to organ damage. Antioxidants can effectively reduce oxidative stress. Vitamin C, vitamin E, lycopene and other studies have shown that traditional antioxidants have a preventive effect on preeclampsia, but in recent years a large sample of randomized trials confirmed that traditional antioxidants can not effectively prevent preeclampsia, and may increase fetal complications disease. This article combines pre-eclampsia