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为了提供金盐对实验性牙周炎作用机理的可能根据,本实验分析比较了应用金盐与未用金盐的实验组与对照组动物(猴)的炎性细胞的特征。当诱发实验性牙周炎二周以后,在有结扎丝的上颌牙颊侧龈取作活检,标本取自上皮层和结缔组织的表层与深层,观察其细胞的特征。接受金盐的实验组标本,其炎性细胞明显少于对照组,主要是多核白细胞,不仅数量减少显著,且在结缔组织内伴有形态变异及退化的成纤维细胞,其它型的炎性细胞则无显著减少或变化。在对照组,则多核白细胞占优势。本文对金盐何以能减少炎性细胞的趋化性和功能作用进行了讨论。根据实验结果,两组保留菌斑的结扎丝
In order to provide a possible basis for the mechanism of action of gold salt on experimental periodontitis, the characteristics of inflammatory cells in experimental and control animals (monkey) using gold salt and non-gold salt were analyzed and compared in this experiment. Two weeks after the experimental periodontitis was induced, a biopsy was performed on the buccal gingival of the maxillary tooth with ligation wire. Specimens were taken from the surface and deep layers of the epithelium and connective tissue to observe the characteristics of the cells. The experimental group received gold salt specimens, the inflammatory cells were significantly less than the control group, mainly polymorphonuclear leukocytes, not only significantly reduce the number, and in the connective tissue accompanied by morphological changes and degeneration of fibroblasts, other types of inflammatory cells There is no significant decrease or change. In the control group, polykaryotic leukocytes predominate. This article discusses why gold salts reduce the chemotaxis and function of inflammatory cells. According to the experimental results, the two groups retain the plaque ligature