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原发性高血压的产生现在多归于遗传、过量摄入钠以及总营养失调等因素。而象矿物质摄入水平这样的调解因素却不曾受人重视。人们早已知道镁盐是致高血压的,认为软水中镁含量低是联系水硬度与正常动脉血压的传递因子。镁对高血压影响的机理尚不清楚,镁是否真能影响血压这一问题也还没确定。现已建立麻醉鼠内脏微血管的血流量和血管直径的测量技术。这一技术也被用于研究限定量镁对鼠血压的影响。重量为100和130 g的Wistar幼雄鼠,分为三组,在12周内给予三种不同的食物。对照鼠
Essential hypertension is now mostly attributed to genetic, excessive intake of sodium and total nausea disorders and other factors. Mediocre factors like mineral intake have not been valued. It has long been known that magnesium salts are hypertensive, suggesting that the low magnesium content in the soft water is a transfer factor that correlates water hardness with normal arterial blood pressure. The mechanism by which magnesium affects hypertension is unclear, and the question of whether magnesium really affects blood pressure has not been established yet. A technique for measuring blood flow and vessel diameter of anesthetized murine visceral microvessels has now been established. This technique has also been used to study the effect of a limited amount of magnesium on rat blood pressure. Wistar young male rats weighing between 100 and 130 g were divided into three groups and given three different foods over 12 weeks. Control mice