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用光镜和电镜研究了6只患有柯萨奇β_3病毒急性心肌炎的C3H/He小鼠的窦房结和房室结组织。6只均有窦房结暂停,其中3只为Ⅱ～Ⅲ°房室传导阻滞。6只小鼠的心脏窦房结和房室结结构明显异常。重要的超微结构变化是特化心肌细胞(Specialized Cells)和神经组织损伤,即窦房结和房室结特化心肌变性、坏死;神经组织的轴突和雪旺氏细胞(Schwann’s Cells)变性,神经肌肉的连接点结构破坏。常见炎症细胞存在于损伤的特化细胞和神经组织中,也见到间质水肿和淋巴管扩张。这些病理
The sinoatrial node and atrioventricular node of C3H / He mice with Coxsackie β_3 virus acute myocarditis were studied by light and electron microscopy. Six had sinus node pause, of which three were Ⅱ ~ Ⅲ ° atrioventricular block. Six of the hearts of the sinoatrial node and atrioventricular node structure abnormalities. Important ultrastructural changes are specificized cells and nerve tissue damage, ie, degeneration and necrosis of the specialized myocardium of the sinus node and atrioventricular node; neurite outgrowth and Schwann’s Cells degeneration , Neuromuscular junction damage structure. Common inflammatory cells are present in damaged specialized cells and nervous tissues, interstitial edema and lymphatic dilation are also seen. These pathologies