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系统性红斑狼疮 (ＳＬＥ)是以Ｔ、Ｂ淋巴细胞功能改变为特征的自身免疫性疾病。ＳＬＥ许多临床症状被认为是Ｂ细胞功能亢进所引起 ,而Ｔ细胞活化是继Ｂ细胞功能亢进、细胞因子分泌、抗体产生、补体活化后的反应[1 ] 。因此 ,Ｔ细胞介导的免疫参与ＳＬＥ病理过程。ＣＤ6 9抗原的
Systemic lupus erythematosus (SLE) is an autoimmune disease characterized by altered T and B lymphocyte function. Many of the clinical symptoms of SLE are thought to be caused by hyperthyroidism of B cells, and T cell activation is a response following hyperthyroidism, cytokine secretion, antibody production, and complement activation [1]. Therefore, T cell mediated immunity is involved in the pathological process of SLE. CD6 9 antigen