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作者对400例卒中患者的回顾性调查发现,25％患者存在某种躯体感觉障碍,2％患者在出现感觉障碍的同时有自发性中枢性卒中后疼痛(CPSP)。躯体感觉障碍的出现意味着脑干躯体感觉通路,中央沟后顶叶皮层和(或)丘脑腹后部与皮层间的联络纤维受累。这些病人的运动障碍常不严重,但可伴有痉挛步态。这可能系病变比引起单纯运动综合征时更靠后所致。CPSP病例中的15％～20％为蛛网膜下腔出血后梗塞而非经典型脑血管病,其中绝大多数(75％)直至卒中后3个月(有时达24个月)才发生疼痛。与无疼痛性躯体感觉障碍的病人相比,疼痛
A retrospective study of 400 stroke patients found that 25% of patients had some somatoform sensorineuria and 2% had spontaneous central stroke pain (CPSP) with sensory disturbances. The presence of somatosensory implication involves the involvement of the somatosensory pathway in the brainstem, the connective fibers between the posterior parietal cortex and (or) the posterior thalamic ventral and the cortex. These patients often dyskinesia, but may be associated with spastic gait. This may be caused by the lesion than when caused by simple motor syndrome. 15% to 20% of CPSP cases are infarctions of subarachnoid hemorrhage rather than classic cerebrovascular disease, with the vast majority of them (75%) developing pain up to 3 months (sometimes up to 24 months) after stroke. Pain compared with patients without painful somatosensory disorders