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近来认为冠状血管痉挛可造成心肌缺血,并且在动脉硬化的发展中起到重要作用。1959年Prinzmetal将ST段抬高的心绞痛命名为变异型心绞痛,并且曾推测冠状动脉痉挛可能是其原因。到了七十年代,随着冠状动脉造影的普及,对冠状动脉痉挛的研究又有进展。现在认为不仅是在变异型心绞痛,就是在伴ST段抬高或下降的劳累型心绞痛、不稳定型心绞痛,甚至在急性心肌梗塞时冠状血管痉挛都是主要的致病因素。能引起冠状血管痉挛的原因有很多,目前有关确切的发病机理尚不清楚。诱发冠状动脉痉挛的方法也很多。如:刺激交感神经的寒冷升压试验,运动负荷试验,拟副交感
Recently, it is thought that coronary vasospasm causes myocardial ischemia and plays an important role in the development of arteriosclerosis. In 1959 Prinzmetal named ST elevation angina pectoris as variant angina pectoris, and it was speculated that coronary spasm may be the reason. In the seventies, with the popularity of coronary angiography, the study of coronary artery spasm has made further progress. It is now argued that coronary vasospasm is a major causative agent not only in variant angina, but also in exertional angina pectoris with elevated or decreased ST-segment elevation, unstable angina, and even in acute myocardial infarction. Can cause coronary vasospasm for many reasons, the current pathogenesis is not yet clear. There are many ways to induce coronary artery spasm. Such as: stimulate the sympathetic cold boost test, exercise load test, to be parasympathetic