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刘承宜等人提出了低强度激光疗法的生物信息模型（ＢＩＭＬ）和激光生物效应的信息转换模型（ＢＩＴＭＬ）。根据ＢＩＭＬ，绿、兰和紫等冷色兴奋Ｇｓ蛋白介导的生理过程；红、橙和黄等暖色兴奋Ｇｑ或Ｇｉ蛋白或受体关联蛋白酶介导的生理过程。根据ＢＩＴＭＬ，在不引起损伤的前提下，一种激光的作用剂量超过一定阈值，它所启动的信使系统将转换成与现有信使系统相拮抗的信使系统。本文应用这些模型讨论低强度激光剂量与其生物效应之间的关系。
Liu Chengyi et al proposed a low-intensity laser bioinformatics model (BIML) and laser bio-effect information conversion model (BITML). Cold-excited Gs-protein-mediated physiological processes, such as BIML, green, blue, and violet; warm-red, orange, and yellow, excited Gq or Gi proteins or receptor-associated protease-mediated physiological processes. According to BITML, without damaging, a laser’s dose exceeds a certain threshold and the messenger system it activates transforms into a messenger system that is antagonistic to the existing messenger system. In this paper, we use these models to discuss the relationship between low-dose laser doses and their biological effects.