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肛隐窝炎是肛瘘的最初状态。早期的研究发现隐窝鳞状上皮和 Morgagni 管柱状上皮的共存区,称为“肛隐窝腺移行区”。该部炎症对深部组织扩散起重要作用。该处肛腺输出管管径正常情况下由1000μm 逐渐变细至40μm。当液性粪便进入后,内括约肌收缩、关闭肛腺输出管,导致瘀滞,发生脓肿,以致形成肛瘘。本研究的目的是证明和区分肛隐窝腺结构的炎症过程,讨论导致形成肛瘘的形态学变化。1979～84年期间治疗96例肛瘘病人,其瘘开口
Anal crypt inflammation is the initial state of anal fistula. Earlier studies found that the coexistence of crypt squamous epithelium and Morgagni tubular epithelium, known as “anal crypt gland transition zone.” Inflammation of the Ministry plays an important role in the proliferation of deep tissues. Where the anal gland output tube diameter under normal circumstances from 1000μm tapered to 40μm. When the liquid into the stool, the contraction of the internal sphincter, closing the anal gland output tube, leading to stasis, abscess, resulting in the formation of anal fistula. The purpose of this study is to demonstrate and differentiate the inflammatory processes of the anal crypts and to discuss the morphological changes that lead to the formation of anal fistulas. Between 1979 and 1984, 96 patients with anal fistula were treated and the fistula was open